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GLP-1 receptor agonists like semaglutide and tirzepatide are making headlines for their weight loss benefits -- but what happens when people start using them without medical oversight? In this episode, we dive into the real risks and unintended consequences of using GLP-1 injections without a doctor's guidance. Ray Solano, Compounding Pharmacist, PDLabs

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Taking GLP-1 medications for weight loss? 🙋‍♀️ Managing your treatment can feel like a lot, but it doesn't have to! Meet Shotwise, your new best friend for simplifying your GLP-1 journey. This app is designed to help you stay organized, consistent, and motivated every step of the way with your GLP-1 treatment. Never miss another dose with our injection scheduler and customizable reminders! Effortlessly track your GLP-1 injections, log dose details, and injection sites to avoid overlaps. Plus, visualize your weight loss progress with our intuitive weight tracker and charts. See how your treatment is working and stay on track! Shotwise is all about making your weight management with GLP-1s hassle-free. Download Shotwise today and take control of your GLP-1 weight loss journey! #GLP1 #WeightLoss #InjectionTracker #Shotwise #WeightManagement #HealthApp #MedicationReminder #GLP1Treatment #WeightLossJourney

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Here is a quick tip on how you should eat before taking your first dose of a GLP-1 medication, such as Ozempic, Wegovy, Trulicity, or Mounjaro, for diabetes or weight loss.

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💉are GLP-1 drugs a metabolic fix or a dangerous shortcut? GLP-1 drugs like Ozempic & Wegovy have taken the weight-loss world by storm, but are they truly mimicking the body's natural processes? While these peptides can be game-changers for metabolic health, they come with critical downsides that few are discussing: ❌ Not a Perfect Replacement – GLP-1 drugs don’t work exactly like the body's natural incretin system, which involves multiple hormones influencing digestion & satiety ❌ Metabolism & Nervous System Impact – These drugs lower heart rate variability (HRV) & reduce sympathetic drive, meaning they may slow metabolic rate in some cases ❌ A Tool, Not a Cure – GLP-1 peptides could be one of the most impactful health innovations in decades, but they aren’t for everyone & should be used strategically Are these drugs truly the future of metabolic health, or are we overlooking serious long-term consequences? Let me know in the comments! 🎧 Tune in to E196 of the Mind Body Peak Performance Podcast with Zack Loewenstein to learn more: ✅ Why GLP-1 Didn’t Work for Zack ✅ Personalized Health Strategies ✅ Discussing Biohacking Tech & Disease Prevention #biohacking #health #glp1 #wegovy #ozempic #ozempicsideeffects #biohackzack #outliyr

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Dang it's hot here in Texas! my video did cut off at the end...I was pretty much done but it was abrupt. What I did not get to was that I think the shipped vial now only contains enough for one month of 20 unit shots instead of a three month supply.

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Click Here to Subscribe: http://Bit.ly/ThomasVid Get MY Groceries at MY Price with Thrive Market: http://ThriveMarket.com/Thomas My Website: http://ThomasDeLauer.com Snacking vs. Fat Loss | The Dangers of Snacking Between Meals | Insulin Spiking- Thomas DeLauer… Snacking - Insulin & Glucagon: Whenever you eat food, your pancreas releases insulin into the blood - as the nutrients are slowly absorbed into cells, insulin levels drop, until finally all the nutrients are absorbed, and insulin levels then remain steady at a low, “baseline” level. So, when you're constantly eating, you're consistently releasing insulin, which puts your body into its "absorptive phase." This means that the insulin in your body is storing sugar and not letting other enzymes in your body release sugar to break down fat. Glucagon: Insulin, the fat-storage and blocking hormone, has a counterpart known as glucagon – a fat-burning and unlocking hormone. About four to six hours after you eat, the glucose levels in your blood decrease, triggering your pancreas to produce glucagon. This hormone signals your liver and muscle cells to change the stored glycogen back into glucose. Glucagon signals the fat cells to release free fatty acids (a process called lipolysis), which signals the body to release stored fat to be used as fuel. Snacking & Digestion: Snacking can affect digestive function as it may increase risk for developing bacterial overgrowth. When bacteria replicate excessively in the small intestine, it results in a condition called Small Intestinal Bacterial Overgrowth. When the body is fasted, the muscles in the small intestine produce a periodic wave of forward motion called the migrating motor complex. The migrating motor complex is a distinct pattern of electromechanical activity observed in gastrointestinal smooth muscle during the periods between meals. It is thought to serve a "housekeeping" role and sweep residual undigested material through the digestive tube. The intestines need to be fasted for at least 90 minutes at a time in order for routine cleansing waves to occur. An increase in gastric, biliary and pancreatic secretion is also seen in conjunction with the motor activity. These secretions aid in the cleansing activity of the migrating motor complex and assist in preventing a buildup of bacterial populations in the proximal segments of the digestive tube. Study - 2 Meals vs 6 Meals per Day: Published in the journal Diabetologica, researchers found that eating 2 larger meals a day (breakfast and lunch) is more effective than 6 smaller meals in a reduced-energy regimen (for patients with type 2 diabetes.) The study compared the effect of six (A6 regimen) vs two meals a day, breakfast and lunch (B2 regimen), on body weight, hepatic fat content (HFC), insulin resistance and beta cell function. Design: Researchers assigned 54 patients with type 2 diabetes, both men and women, age 30–70 years, to follow two regimens of a hypoenergetic diet (below normal, caloric restriction), A6 and B2, each for 12 weeks. The diet in both regimens had the same macronutrient and energy content. Results: Body weight decreased in both regimens, more for B2 (-5.07 lbs for A6 vs −8.1 lbs for B2) HFC decreased in response to both regimens, more for B2 (−0.03% for A6 vs −0.04% for B2) Fasting plasma glucose and C-peptide levels decreased in both regimens, more for B2 Fasting plasma glucagon decreased with the B2 regimen, whereas it increased for the A6 regimen OGIS (oral glucose insulin sensitivity) increased in both regimens, more for B2 - No adverse events were observed for either regimen. Conclusion: Eating only breakfast and lunch reduced body weight, HFC, fasting plasma glucose, C-peptide and glucagon, and increased OGIS, more than the same caloric restriction split into six meals. References: 1) How Insulin Really Works: It Causes Fat Storage...But Doesn't Make You Fat | Muscle For Life. (n.d.). Retrieved from https://www.muscleforlife.com/how-insulin-works/ 2) Understanding Our Bodies: Insulin | Nutrition Wonderland. (n.d.). Retrieved from http://nutritionwonderland.com/2010/05/understanding-our-bodies-insulin/ 3) Insulin and Glucagon: How Do They Work? (n.d.). Retrieved from http://www.healthline.com/health/diabetes/insulin-and-glucagon#Workingtogether2 4) Unlock Glucagon: Your Body's Fat-Burning Hormone. (n.d.). Retrieved from http://healinggourmet.com/weight-loss-diet/unlock-glucagon-fat-burning-hormone/ 5) The effect of meal frequency in a reduced-energy regimen on the gastrointestinal and appetite hormones in patients with type 2 diabetes: A randomised crossover study. (n.d.). Retrieved from http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0174820 6) The Migrating Motor Complex. (n.d.). Retrieved from http://www.vivo.colostate.edu/hbooks/pathphys/digestion/stomach/mmcomplex.html

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